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French researchers to give nicotine patches to coronavirus patients, frontline workers after low infection rates found among smokers

Mary Kekatos
Daily Mail, UK
Thu, 23 Apr 2020 15:12 UTC

Comment: They would save themselves trouble if they just gave them tobacco to smoke...


Man with nicotine patch
French researchers are planning to trial whether nicotine patches will help prevent - or lessen the effects of - the deadly coronavirus.

Evidence is beginning to show the proportion of smokers infected with coronavirus is much lower than the rates in the general population.

Scientists are now questioning whether nicotine could stop the virus from infecting cells, or if it may prevent the immune system overreacting to the infection.

Doctors at a major hospital in Paris - who also found low rates of smoking among the infected - are now planning to give nicotine patches to COVID-19 patients.

They will also give them to frontline workers to see if the stimulant has any effect on preventing the spread of the virus, according to reports.

Comment: BUT they're only giving them to select COVID-19 patients for 'trials'. Meanwhile, they're moving to BAN the sale of nicotine patches. They know tobacco products work, so they're in a race to control the supply before people realize the scam:

France bans online sales of nicotine products, limits sale in pharmacies

See also:

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Corona

Coronavirus Lies: Babies And Children Are NOT 'Dying From COVID-19'

Juliana Barembuem
Sott.net
Thu, 23 Apr 2020 13:29 UTC
covid-19 1984

A US protester against the lockdown
The use of language - or rather, the misuse of language - remains at the forefront of the media's effort to maintain global public support for the most draconian and widespread curtailment of civil liberties ever implemented.

The most successful lies are always false representations of reality that are based on something true, and they typically appeal to our better instincts. But if you don't read between the lines, the liars win, and you will end up doing the opposite to what you believe you are doing - in this case, protecting the vulnerable and your loved ones.

After it became apparent that COVID-19 broadly afflicts only the very old and very sick, a media blitz followed about tragic cases of COVID-19 in minors and infants. In the following video I analyze some of these reports and show them to be, at best, without foundation - at worst, insidious lies by omission.
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SOTT Logo Radio

Objective:Health - Thriving and Surviving During the Corona Plandemic


Sott.net
Wed, 22 Apr 2020 00:00 UTC
O:H header
The consequences of the coronavirus lockdown are dire from whatever perspective you look. The entire world economy has been all but shut down for the past few months, leaving people in dire straights on every level. The implications are huge, with some citing comparisons to the Great Depression.

On this episode of Objective:Health, we're joined by Mike the Bear who has been researching and working in financial markets for 20 years. His insights on the market, supply chains and cryptocurrencies put him in a unique position to comment on the current state of things and possible implications for the future.

But it's not all doom and gloom. Mike gives us some good advice on what we can do to help protect ourselves in the current climate, giving practical tips for everyone, even if you're not a market genius.

Join us for a scintillating discussion about what you can do to stay secure in insecure times.


And check us out on Brighteon!


Running Time: 01:05:17

Download: MP3 — 59.5 MB

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Syringe

'I have the right to express my views': Djokovic defends 'anti-vaxxer' stance after Covid-19 comments cause stir


RT
Tue, 21 Apr 2020 19:00 UTC
Tennis star Novak Djokovic
Tennis star Novak Djokovic has defended comments he made in relation to a potential vaccine for the novel coronavirus, saying that he is against mandatory vaccinations for the virus in order to be able to travel.

Djokovic prompted something of a media frenzy this past weekend when he highlighted his concerns about potentially being 'forced' to receive a vaccination for coronavirus - despite this possibly leading to difficulties in traveling the tennis circuit.

Predictably, Djokovic's comments were questioned by science experts along with some of his fellow professionals, who maintain that vaccination is a necessary step required to halt the spread of the deadly virus.

Comment: It's quite telling that anyone expressing any need for caution or right to choose in regards to the vaccine issue is labelled an 'anti-vaxxer', regardless of how nuanced their position is. Trying to argue with an authoritarian is always doomed to fail. You're either with them, or you're against them.

See also:

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Microscope 2

Coronavirus now has thousands of strains and mutations, some rare, some more pathogenic

Stephen Chen
South China Morning Post
Mon, 20 Apr 2020 10:41 UTC
coronavirus
© EPA-EFE
A team led by Professor Li Lanjuan has studied how the novel coronavirus mutates and possible implications for the pandemic.
A new study by one of China's top scientists has found the ability of the new coronavirus to mutate has been vastly underestimated and different strains may account for different impacts of the disease in various parts of the world.

Professor Li Lanjuan and her colleagues from Zhejiang University found within a small pool of patients many mutations not previously reported. These mutations included changes so rare that scientists had never considered they might occur.

They also confirmed for the first time with laboratory evidence that certain mutations could create strains deadlier than others.

"Sars-CoV-2 has acquired mutations capable of substantially changing its pathogenicity," Li and her collaborators wrote in a non-peer reviewed paper released on preprint service medRxiv.org on Sunday.

Comment: More detail on the strains from meaww.com:
"We provide direct evidence that the SARS-CoV-2 has acquired mutations capable of substantially changing its pathogenicity. Our study provides direct evidence that mutations currently occurring in the SARS-CoV-2 genome have the functional potential to impact the viral pathogenicity," says the team in their findings, a pre-print version of which has been published.

"A diverse collection of mutations was identified in the 11 viral isolates, including two sets of founding mutations for two major clusters of viruses currently infecting the world population. In addition, 19 identified mutations are novel, despite the relatively early sampling dates, indicating that the true diversity of the viral strains is still largely underappreciated," they say.

In a previous study, researchers from China had identified two separate strains of the new coronavirus. The research team analyzed 103 publicly available genomes from infected persons and found 70% were the more aggressive type, while the remaining 30% comprised the less aggressive type. Another study discovered three distinct strains of COVID-19, consisting of clusters of "closely related lineages" — which have been labeled 'A', 'B' and 'C'.
Despite evidence to the contrary and even acknowledging that the exact origin is not yet possible to determine, the WHO is still pushing the animal to human transmission theory. Meanwhile Trump is looking into the possibility it originated from a lab, albeit in Wuhan and not the US:
Asked to comment on recent reports by major US media outlets suggesting the lethal virus emanated from a virology lab in the Chinese city of Wuhan, Takeshi Kasai, WHO regional director for the Western Pacific, cautioned against speculation, saying the verdict was still out on the exact origins of the pathogen.

"I think at this stage it's not possible to determine the precise source, but available evidence suggests that it has an origin from animals," Kasai said at a virtual press conference on Tuesday. "Again, there are many researchers studying these issues, and we've heard that this might have originated from bats, but how it reached humans we still don't know."

"At this time, we cannot conclude anything yet."

Last week, Fox News and the Washington Post both ran stories, largely based on anonymous US government sources, suggesting that the coronavirus might have escaped from the lab in Wuhan. Although US President Donald Trump fell short of endorsing the theory outright, he threw his weight behind the idea, saying it "seems to make sense," and that the White House was now looking into the question.

Not everybody on Trump's own Covid-19 task force seems to be on board, however. One key figure, Dr Anthony Fauci, has been dismissive of the claim, insisting at a recent briefing that the virus was not "man-made" and had instead evolved naturally to infect humans.

As drama and speculation about the pandemic swirl in the political realm, however, scientists have been hard at work investigating the virus' origins, with one recent study led by geneticists at Cambridge University concluding that it may have come on the scene much earlier than previously thought. While a number of experts have estimated that the first human infection occurred sometime in November or December 2019 near a wildlife market in Wuhan, the Cambridge researchers believe the outbreak actually started weeks prior, in September, and it may not have happened in Wuhan at all, but somewhere south of the central Chinese city. While the geneticists could provide few definite answers, the mystery may be unraveled further as research continues, with scientists building genetic models to trace the virus back to its true point of origin.
See also:

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Health

Fauci backpedals on vitamin C and D recommendations

Dr. Joseph Mercola
Mercola.com
Wed, 22 Apr 2020 00:01 UTC
Dr. Anthony Fauci
As reported in my April 7, 2020, article "Vitamins C and D Finally Adopted as Coronavirus Treatment," the largest hospital system in New York, Northwell Health, has been giving COVID-19 patients admitted into intensive care 1,500 milligrams of intravenous vitamin C three to four times a day in conjunction with the antimalarial drug hydroxychloroquine and the antibiotic azithromycin, both of which have shown promise in coronavirus treatment.1

According to Dr. Andrew G. Weber, a pulmonologist and critical-care specialist affiliated with two Northwell Health facilities on Long Island, "The patients who received vitamin C did significantly better than those who did not get vitamin C. It helps a tremendous amount, but it is not highlighted because it's not a sexy drug."2

Comment: See also:

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Bad Guys

The Gates Of Hell: PROVEN - Coronavirus vaccines CAUSED lung inflammation and death

Joaquin Flores
Fort Russ
Mon, 20 Apr 2020 17:36 UTC
bill gates
© DR
From The Children's Health Defense Team:
At first, the experiment seemed successful as all the animals developed a robust antibody response to coronavirus. However, when the scientists exposed the vaccinated animals to the wild virus, the results were horrifying. Vaccinated animals suffered hyper-immune responses including inflammation throughout their bodies, especially in their lungs. Researchers had seen this same "enhanced immune response" during human testing of the failed RSV vaccine tests in the 1960s. Two children died.
2012 Study: Immunization with SARS coronavirus vaccines leads to pulmonary immunopathology on challenge with the SARS virus.

"However, challenge of mice given any of the vaccines led to occurrence of Th2-type immunopathology suggesting hypersensitivity to SARS-CoV components was induced. Caution in proceeding to application of a SARS-CoV vaccine in humans is indicated." [emphasis our own - ed]

Why are the world's top vaccine promoters, like Paul Offit and Peter Hotez, frantically warning us about the unique and frightening dangers inherent in developing a coronavirus vaccine?

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Attention

Autopsies reveal COVID-19 attacks blood vessels, causes multiple organ failure in some patients - study


Sputnik
Tue, 21 Apr 2020 17:23 UTC
Covid-19 patient
© REUTERS / Lucas Jackson
The SARS-CoV-2 coronavirus resulted in more severe illnesses than pneumonia, researchers from the University Hospital Zurich wrote in a study published last week.

COVID-19, the disease responsible for the ongoing coronavirus pandemic, can cause multiple organ failure after attacking the lining of blood vessels in the body, it was revealed in a Lancet study.

The findings show that smokers and those with pre-existing conditions, namely obesity and cardiovascular disorders such as hypertension, were affected most when catching the virus.

Comment: Looks like the medical establishment had to go into overdrive to counteract the mainstream reporting that smoking actually has provided protection from the potential damage caused by the virus. See:

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Sun

Sunlight destroys coronavirus quickly, new US Government tests find

Jana Winter and Sharon Weinberger
Yahoo! News
Fri, 17 Apr 2020 00:00 UTC
Lab experiments show that the coronavirus does not survive long in high temperatures and high humidity
CoronaVirus
© Getty Images
Coronavirus - Photo illustration
Preliminary results from government lab experiments show that the coronavirus does not survive long in high temperatures and high humidity, and is quickly destroyed by sunlight, providing evidence from controlled tests of what scientists believed — but had not yet proved — to be true.

A briefing on the preliminary results, marked for official use only and obtained by Yahoo News, offers hope that summertime may offer conditions less hospitable for the virus, though experts caution it will by no means eliminate, or even necessarily decrease, new cases of COVID-19, the disease caused by the coronavirus. The results, however, do add an important piece of knowledge that the White House's science advisers have been seeking as they scramble to respond to the spreading pandemic.

The study found that the risk of "transmission from surfaces outdoors is lower during daylight" and under higher temperature and humidity conditions. "Sunlight destroys the virus quickly," reads the briefing.
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Life Preserver

COVID-19, ACE2, Nicotinic Receptors And The Cholinergic Anti-Inflammatory And Cognitive-Improving Pathway

Dr.Gaby
Sott.net
Sun, 19 Apr 2020 21:07 UTC
COVID-19 ACE2
© Getty Images/selvanegra
Graphic representation of COVID-19 binding to the ACE2 receptor on human cells via its spike protein (red)
I've been reading case reports of heart damage in people infected with COVID-19. In fact, someone in my close circle had an episode of fulminant myopericarditis and a strange lung pathology last December, from which he miraculously recovered. I deem SARS-CoV-2 (the virus that causes the disease COVID-19) a likely suspect behind my friend's illness, even though this occurred a few months before the pandemic officially began in Europe.

The following LiveScience article provides some clues as to how both the heart and lungs can be seriously affected during a COVID-19 infection:
The mysterious connection between the coronavirus and the heart

"We're seeing cases of people who don't have an underlying heart disease, who are getting heart damage," said Dr. Erin Michos, the associate director of preventive cardiology at Johns Hopkins School of Medicine. Heart damage isn't typical in mild cases of COVID-19, and tends to occur more often in patients who have severe symptoms and are hospitalized, she said. [...]

Both heart cells and lung cells are covered with surface proteins known as angiotensin-converting enzyme 2 (ACE2) — these molecules serve as "doorways" for the virus to enter cells. But this enzyme is a "double-edged sword," she said. On one hand, the ACE2 molecule acts as a gateway for the virus to enter the cell and replicate, but on the other hand, it normally serves a "protective" function, Michos said.

When tissues in the body are damaged — either by an invading virus such as SARS-CoV-2 or by other means, the body's natural healing response involves releasing inflammatory molecules, such as small proteins called cytokines, into the bloodstream. But paradoxically, too much inflammation can actually make things worse. The ACE2 enzyme acts as an anti-inflammatory, keeping immune cells from inflicting more damage on the body's own cells.

But when the virus latches onto ACE2 proteins, these proteins get knocked out of commission, possibly reducing the anti-inflammatory protection that they give. So the virus may be acting as a double-whammy by damaging cells directly and preventing the body from protecting tissues from inflammatory damage.

"If the heart muscle is inflamed and damaged by the virus, the heart can't function," she said.

The novel coronavirus might also indirectly damage the heart. In this scenario, the patient's immune system winds up "going haywire," Michos said. This scenario has played out in some really sick patients who have highly elevated inflammatory markers — or proteins that signal high levels of inflammation in the body.

This is called a "cytokine storm," Michos said. Cytokine storms damage organs throughout the body, including the heart and liver, she added. It's not clear why some people have such an elevated response compared with others, but some people could be genetically prone to it, she added. [...]

It's really hard to tease out whether having more ACE2 is helpful or harmful, as these proteins are how the virus enters the cells, but also known to protect the cells against injury, Michos said.
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